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Studies consistently show altered MGBA composition in AD patients, suggesting a possible role in disease onset and progression, as shown in Table 1

These include regulation of mechanisms controlling reactive oxygen species (ROS), which cause macromolecular damage 16,17 , mitochondrial turnover or mitophagy 18 , and the mitochondrial unfolded protein response (UPR mt ), in which mitochondrial damage can be prevented by the removal of aggregated or unfolded proteins 19

DOI: 10.2147/CCID.S128339

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